Ray wrote in many of is articles that menopause is not what mainstream medicine describes it to be. There is no “fixed” number of eggs predetermined in-utero before the female baby is even born. Eggs can be produced at any point of time in a female organism’s life, provided the metabolic state of the organism is optimal. In addition, there is also no structural ovarian failure in menopause, despite medicine claims to the contrary. The study below now officially confirms Ray’s writings, and suggests that “menopause” is simply the female version of the so-called age-related male hypogonadism, which is known to be entirely of secondary (hormonal) origin in most men and reversible by administration of estrogen “antagonists” (risky and more accurately called SERM) such as clomiphene/clomid or chemicals that increase mitochondrial cholesterol transport for steroid synthesis by activating the StAR protein, or simply by administering testosterone therapy. Btw, aside from the testosterone therapy, the same approach has already been shown to work in female too, but I don’t think SERMs are safe in either sex. In any event, it seems that just as there is no sign of structural damage in aging male gonads, there seems to be no such damage in female gonads (ovaries) either. In addition, human oocytes appear to also preserve their energy production, despite advancing age. The study does actually make the claim that female child-bearing age can be extended and perhaps maintained throughout the entire lifespan. Ray suggested progesterone, thyroid, aspirin and vitamin A for menopause. More recent studies have demonstrated that DHEA, vitamin D, and vitamin E may also help with restoring proper ovarian function to youthful levels.
https://pubmed.ncbi.nlm.nih.gov/40768572/
“…A NEW study has found that human egg cells remain genetically stable as women age, resisting the accumulation of mutations in their mitochondrial DNA. The findings suggest that oocytes possess unique protective mechanisms that preserve mitochondrial integrity, helping maintain reproductive potential even later in life. Researchers led by Barbara Arbeithuber and Kateryna Makova used high-precision duplex sequencing to analyse mitochondrial DNA (mtDNA) mutations in single oocytes, blood, and saliva samples from women aged 20 to 42. They discovered that while mutations increased with age in blood and saliva, no such rise occurred in oocytes. This indicates that human eggs, unlike most body cells, are shielded from age-related mitochondrial mutations—a finding that could reshape understanding of reproductive aging. Further analysis showed that high-frequency mutations were less common in coding regions of mtDNA, suggesting that purifying selection removes potentially harmful variants, while lower-frequency mutations appeared evenly distributed. “These results highlight the resilience of human oocytes,” said the authors. “Their mitochondria appear protected from the accumulation of age-related mutations, preserving energy production and genetic integrity.” The study offers reassuring news as more women choose to conceive later in life. By showing that oocyte mitochondria remain robust over time, it deepens scientific insight into fertility longevity and may inform future advances in reproductive medicine.”
