As many of my readers know, muscle spasticity is one of the main symptoms of low thyroid function and is present in many people with chronic conditions, especially neurological (e.g. multiple sclerosis). A number of remedies have been tried clinically, but there has been no breakthrough, except with quinine. However, for some reason, medicine has decided that quinine is “dangerous” and its use is discouraged clinically while also being regulated in the food supply. Tonic water, which traditionally has 300mg-500mg quinine per quart\/liter, has been regulated in most Western countries to contain no more than 100mg per quart\/liter. Be that as it may, I think the focus should stay on the fact that impaired muscle relaxation is core symptom of energetic deficiency. As an extreme example, Ray cited experiments from the early 20th century demonstrating that even “rigor mortis” can be prevented with injections of ATP directly into the muscle tissue. The study below found that just 180mcg vitamin K2 (MK-7) daily, for 8 weeks strongly decreased frequency and severity of nocturnal leg\/muscle cramps (NLC), while also producing no notable side effects. In addition, the study cites a previous one where vitamin K was able to reduce muscle cramps in hemodialysis patients. The study attributes these effects of vitamin K to its ability to inhibit calcium uptake into the cell from the extracellular medium. I would take this a step further and point out that vitamin K can even reverse already established soft-tissue calcification, which is the reverse process – i.e. pushing calcium out of the cell after it had already accumulated in pathological amounts. However, I think the main mechanism of vitamin K has to be related to its ability to act as a quinone\/oxidizer and speed up the flow of electrons through the OXPHOS pathway. In corroboration, T3 administered orally or by injection also has potent muscle-relaxing effects and T3 can also decalcify tissues by raising ATP production (and thus magnesium retention) and accelerating calcium efflux by raising CO2 production (which draws calcium out of the cell as CO2 is leaving the cell) thus achieving higher magnesium\/calcium ratio inside the cell. Vitamin K also has this effect and this is probably how it also manages to decalcify cells (an effect already demonstrated in several human trials).<\/p>\n
https:\/\/jamanetwork.com\/journals\/jamainternalmedicine\/fullarticle\/2825457<\/a><\/p>\n